APOA1 and VKORC1

  • Number of citations of the paper that reports this interaction (PMID 21103663)
  • 2
  • Data Source:
  • BioGRID (two hybrid)

APOA1

VKORC1

Gene Name apolipoprotein A-I vitamin K epoxide reductase complex, subunit 1
Image No pdb structure
Gene Ontology Annotations Cellular Component
Molecular Function
Biological Process
Pathways
Drugs
Diseases
GWAS
Protein-Protein Interactions 35 interactors: ABCA1 ABCA13 AKT2 APOA1BP APOA2 APOB APOC1 APOF APOL1 APP ATP5B CETP CFHR1 CLEC4G CMTM5 FBLN1 GPLD1 HPR JAK3 LBP LCAT MAP7D1 MAPK6 PCMT1 PDE1A PLTP PON1 SAA2 SCARB1 SPP1 TNFRSF10C TOMM20 TTR VKORC1 ZNRD1 80 interactors: AHSG APLP2 APOA1 APOA2 APOC1 APOC3 APOC4 ARL6IP5 ASGR1 ASGR2 ATP5G3 ATP6AP2 BCAP31 C1S CCDC12 CCDC167 CD63 CD81 CD9 CLDN10 CNN3 COMT CXADR DPM2 ECHDC1 FGG FGL1 FIS1 FTL GJB1 HAMP HDAC6 HOMER2 HP HPR HSD17B7 IFITM3 JAGN1 KCNK17 LIG4 METTL7B MSMO1 MSRB1 MT1X PCDH1 PCMT1 PDIA6 PIGH PLP2 PPA2 PPIB PROCR REEP5 RMDN1 RMND5A SAA1 SAT1 SERPINA1 SLC15A3 SLC25A1 SLC31A2 SNORA12 SPTSSA STAT6 SURF4 TF TMBIM4 TMEM120A TMEM134 TMEM176A TMEM230 TMEM242 TMEM256 TMEM45B TRAM1 TREX1 VAPB YIF1A ZCCHC24 ZMYM6NB
Entrez ID 335 79001
HPRD ID 02517 10540
Ensembl ID ENSG00000118137 ENSG00000167397
Uniprot IDs P02647 Q9BQB6
PDB IDs 1AV1 1GW3 1GW4 1ODP 1ODQ 1ODR 2A01 3J00 3K2S 3R2P
Enriched GO Terms of Interacting Partners?
Tagcloud ?
admission  atherosclerosis  believed  concentration  consequence  controls  explain  explanation  explicable  fifth  genotypes  hdl  immediately  infarction  mi  microg  min  ml  myocardial  nmol  occured  paraoxonase  pon1  reside  risen  subfraction  subgroups  thrombolytic  variation 
arg98trp  arginine  carboxylase  carboxylated  carboxylation  catalyzes  clotting  colocalization  confocal  consequence  di  disrupts  episodes  epoxide  exits  hydroquinone  pathophysiology  reductase  restores  silico  suffer  supplementation  supplemented  supraphysiological  unrelated  vitamin  vkcfd2  vkd 
Tagcloud (Difference) ?
admission  atherosclerosis  believed  concentration  controls  explain  explanation  explicable  fifth  genotypes  hdl  immediately  infarction  mi  microg  min  ml  myocardial  nmol  occured  paraoxonase  pon1  reside  risen  subfraction  subgroups  thrombolytic  variation 
arg98trp  arginine  carboxylase  carboxylated  carboxylation  catalyzes  clotting  colocalization  confocal  di  disrupts  episodes  epoxide  exits  hydroquinone  pathophysiology  reductase  restores  silico  suffer  supplementation  supplemented  supraphysiological  unrelated  vitamin  vkcfd2  vkd 
Tagcloud (Intersection) ?
consequence