TRAF5 and RIPK1

  • Number of citations of the paper that reports this interaction (PMID 11479302)
  • 66
  • Data Source:
  • HPRD (in vivo)

TRAF5

RIPK1

Gene Name TNF receptor-associated factor 5 receptor (TNFRSF)-interacting serine-threonine kinase 1
Image No pdb structure
Gene Ontology Annotations Cellular Component
Molecular Function
Biological Process
Pathways
Drugs
Diseases
GWAS
Protein-Protein Interactions 42 interactors: CAPNS1 CCDC184 CD27 CD40 COG7 COMMD3 DGKH ENOX2 GORASP2 LTBR MAP3K14 MAP3K5 MAVS MDM2 NGFRAP1 NMI NUMBL RAD23A RIPK1 RIPK2 RNF111 SDCBP SEC13 SERPINB9 TDP2 THAP1 TNFRSF11A TNFRSF12A TNFRSF13B TNFRSF14 TNFRSF19 TNFRSF4 TNFRSF8 TRAF2 TRAF3 TRAF3IP2 TRAF6 TRIM27 TRIM37 TRIM8 USP7 ZFAND6 45 interactors: BIRC2 BIRC3 CASP8 CCDC50 CDC42BPB CFLAR CRADD EGFR FADD FAS HSP90AA1 IKBKB IKBKG ITCH MAP3K14 MAP3K3 MAP3K8 NFKB1 NUMBL OTUD1 PELI1 PTK2 RIPK3 RNF11 RNF216 SQSTM1 TAX1BP1 TICAM1 TNF TNFAIP3 TNFRSF10A TNFRSF10B TNFRSF1A TRADD TRAF1 TRAF2 TRAF3 TRAF5 TRIM38 TRPC4AP UBE2N USP2 XIAP YOD1 ZFAND5
Entrez ID 7188 8737
HPRD ID 03834 04583
Ensembl ID ENSG00000082512 ENSG00000137275
Uniprot IDs D3DT93 O00463 Q13546
PDB IDs 4ITH 4ITI 4ITJ
Enriched GO Terms of Interacting Partners?
Tagcloud ?
adhesion  affinity  bind  cd40  cd40ct  cytoplasmic  dispensable  dominantly  extensive  icam  ikappabalpha  implicated  incapable  intercellular  jak3  kappab  mediators  molecule  much  mutants  necrosis  residues  strongly  tail  traf2  traf3  traf6  transduction 
allowed  autocrine  back  deficient  endoplasmic  er  instead  interacting  intrinsic  kills  l929  lineage  lymphotoxin  mlkl  necroptosis  necrosis  opportunities  pathologies  protect  remarkably  repression  rescue  reticulum  ripk3  stress  stressors  switch  tnfr1  unresolved 
Tagcloud (Difference) ?
adhesion  affinity  bind  cd40  cd40ct  cytoplasmic  dispensable  dominantly  extensive  icam  ikappabalpha  implicated  incapable  intercellular  jak3  kappab  mediators  molecule  much  mutants  residues  strongly  tail  traf2  traf3  traf6  transduction 
allowed  autocrine  back  deficient  endoplasmic  er  instead  interacting  intrinsic  kills  l929  lineage  lymphotoxin  mlkl  necroptosis  opportunities  pathologies  protect  remarkably  repression  rescue  reticulum  ripk3  stress  stressors  switch  tnfr1  unresolved 
Tagcloud (Intersection) ?
necrosis