TRAF3 and TNFRSF14

  • Number of citations of the paper that reports this interaction (PMID 9162022)
  • 62
  • Data Source:
  • HPRD (in vivo)

TRAF3

TNFRSF14

Gene Name TNF receptor-associated factor 3 tumor necrosis factor receptor superfamily, member 14
Image
Gene Ontology Annotations Cellular Component
Molecular Function
Biological Process
Pathways
Drugs
Diseases
GWAS
  • Brain imaging in schizophrenia (interaction) ( 19023125)
Protein-Protein Interactions 55 interactors: ABCC1 ALPL APOBEC3C AZI2 BIRC3 CASP3 CD27 CD40 DAXX EDA2R EDAR EWSR1 FAS GRIN1 GRIN2D IL15 IRF1 LTBR MAP2K1 MAP3K14 NGFRAP1 NUP62 OTUD5 PDLIM7 RIPK1 RRAS2 SAR1A SRC TANK TBK1 TDP2 TICAM1 TNFRSF11A TNFRSF12A TNFRSF13C TNFRSF14 TNFRSF17 TNFRSF18 TNFRSF19 TNFRSF1A TNFRSF1B TNFRSF4 TNFRSF8 TNFRSF9 TRADD TRAF2 TRAF3IP1 TRAF3IP2 TRAF3IP3 TRAF5 TRIM37 UBE2I USP7 ZBP1 ZMYND11 30 interactors: A2M APP BTLA CD160 CHD3 DGKD DRAP1 DYNLL1 ESR1 HDAC3 HDAC4 IMPA2 ITFG1 KIAA1377 LTA MBTPS1 NDUFS2 NRIP1 RAD21 SERPINA4 TNFSF13 TNFSF14 TRAF1 TRAF2 TRAF3 TRAF5 UBQLN4 VIM WDR73 ZBTB48
Entrez ID 7187 8764
HPRD ID 03539 04122
Ensembl ID ENSG00000131323 ENSG00000157873
Uniprot IDs A6NHG8 Q13114 Q92956
PDB IDs 1FLK 1FLL 1KZZ 1L0A 1RF3 1ZMS 2ECY 2GKW 1JMA 2AW2 4FHQ
Enriched GO Terms of Interacting Partners?
Tagcloud ?
adhesion  affinity  bind  cd40  cd40ct  cytoplasmic  dispensable  dominantly  extensive  icam  ikappabalpha  implicated  incapable  intercellular  jak3  kappab  mediators  molecule  much  mutants  necrosis  residues  strongly  tail  traf2  traf5  traf6  transduction 
aberrations  additional  advances  approaches  biological  closely  contribute  crebbp  ep300  epha7  expected  ezh2  fl  follicular  genetic  hallmark  hand  individuals  lymphoma  mef2b  microenvironment  mll2  pathogenesis  recognized  recurrent  technology  translocation  understanding  unique 
Tagcloud (Difference) ?
adhesion  affinity  bind  cd40  cd40ct  cytoplasmic  dispensable  dominantly  extensive  icam  ikappabalpha  implicated  incapable  intercellular  jak3  kappab  mediators  molecule  much  mutants  necrosis  residues  strongly  tail  traf2  traf5  traf6  transduction 
aberrations  additional  advances  approaches  biological  closely  contribute  crebbp  ep300  epha7  expected  ezh2  fl  follicular  genetic  hallmark  hand  individuals  lymphoma  mef2b  microenvironment  mll2  pathogenesis  recognized  recurrent  technology  translocation  understanding  unique 
Tagcloud (Intersection) ?