HMGB1 and CUX1

  • Number of citations of the paper that reports this interaction (PMID 11748221)
  • 27
  • Data Source:
  • HPRD (in vitro)

HMGB1

CUX1

Gene Name high mobility group box 1 cut-like homeobox 1
Image No pdb structure
Gene Ontology Annotations Cellular Component
Molecular Function
Biological Process
Pathways
Drugs
Diseases
GWAS
  • Response to antidepressants ( 22584459)
Protein-Protein Interactions 73 interactors: AES AGER AGTRAP AR ATOH1 C14orf1 CASP3 CDK1 CEBPB CREBBP CRMP1 CSNK1A1 CTCF CUX1 DNM2 DNMT1 ERF FOXA3 FOXC1 GTF2A1 HES1 HMGA1 HNRNPK HOXB1 HOXB3 HOXC6 HOXD10 HOXD11 HOXD3 HOXD8 HOXD9 HR IRF2 LRIF1 MECP2 MNT NCAN NEUROD6 NFKB1 NR3C1 PGR PLAT PLG POU5F1 PPP2R3A PRKCA PRKDC PSEN1 PTPRZ1 RAD23B RAG1 RASSF4 RB1 RBPJ RELA RFX1 SIX5 SOX18 TAF1 TBP TERF2 TERF2IP TFE3 TGIF1 TLE1 TLE2 TLR2 TLR4 TP53 TP73 UNC119 ZFP36 ZNF24 20 interactors: BIN1 CA12 CCNA1 CDC25A CDK1 CDK7 CREBBP CTSL CYTH2 EHMT2 ELAC2 GOLGA5 HMGB1 KAT2B MAX RB1 RECQL5 SATB1 SDC3 TLE4
Entrez ID 3146 1523
HPRD ID 01228 00295
Ensembl ID ENSG00000189403 ENSG00000257923
Uniprot IDs P09429 Q5T7C4 P39880 Q13948 Q3LIA3
PDB IDs 2LY4 2YRQ
Enriched GO Terms of Interacting Partners?
Tagcloud ?
1racp  abilities  analysed  ankylosing  asdas  basdai  basfi  bath  besides  box  chromosomal  compare  correlations  crp  elisa  erythrocytesedimentationrate  esr  from71  healthy  index  mobility  pbmcs  rage  remarkably  spondylitis  surface  tlr2  tlr4  untreated 
accumulated  activators  ancillary  base  burden  cut  ectopic  excision  framework  haploinsufficiency  homeobox  implicated  isoform  isoforms  mechanistic  opposite  paradoxically  progression  promotes  protection  reconcile  repressors  supports  suppression  transgenic  tumorigenesis  tumour  whereby  yet 
Tagcloud (Difference) ?
1racp  abilities  analysed  ankylosing  asdas  basdai  basfi  bath  besides  box  chromosomal  compare  correlations  crp  elisa  erythrocytesedimentationrate  esr  from71  healthy  index  mobility  pbmcs  rage  remarkably  spondylitis  surface  tlr2  tlr4  untreated 
accumulated  activators  ancillary  base  burden  cut  ectopic  excision  framework  haploinsufficiency  homeobox  implicated  isoform  isoforms  mechanistic  opposite  paradoxically  progression  promotes  protection  reconcile  repressors  supports  suppression  transgenic  tumorigenesis  tumour  whereby  yet 
Tagcloud (Intersection) ?