SAA2 and APOA1

  • Number of citations of the paper that reports this interaction (PMID 11991719)
  • 11
  • Data Source:
  • BioGRID (two hybrid)

SAA2

APOA1

Gene Name serum amyloid A2 apolipoprotein A-I
Image No pdb structure
Gene Ontology Annotations Cellular Component
Molecular Function
Biological Process
Pathways
Drugs
Diseases
GWAS
Protein-Protein Interactions 3 interactors: APOA1 COL4A1 LAMA1 35 interactors: ABCA1 ABCA13 AKT2 APOA1BP APOA2 APOB APOC1 APOF APOL1 APP ATP5B CETP CFHR1 CLEC4G CMTM5 FBLN1 GPLD1 HPR JAK3 LBP LCAT MAP7D1 MAPK6 PCMT1 PDE1A PLTP PON1 SAA2 SCARB1 SPP1 TNFRSF10C TOMM20 TTR VKORC1 ZNRD1
Entrez ID 6289 335
HPRD ID 00098 02517
Ensembl ID ENSG00000134339 ENSG00000118137
Uniprot IDs P0DJI9 P02647
PDB IDs 1AV1 1GW3 1GW4 1ODP 1ODQ 1ODR 2A01 3J00 3K2S 3R2P
Enriched GO Terms of Interacting Partners?
Tagcloud ?
10scn  1rn  alleles  amyloidosis  c57bl  chemotaxis  del  deposit  differently  efficacious  encoded  equally  familiar  fever  formyl  fpr2  isoforms  mediterranean  mobilization  modulating  predominant  preference  robust  saa1  selectivity  tlr2  tlr4lps  toll 
admission  atherosclerosis  believed  concentration  consequence  controls  explain  explanation  explicable  fifth  genotypes  hdl  immediately  infarction  mi  microg  min  ml  myocardial  nmol  occured  paraoxonase  pon1  reside  risen  subfraction  subgroups  thrombolytic  variation 
Tagcloud (Difference) ?
10scn  1rn  alleles  amyloidosis  c57bl  chemotaxis  del  deposit  differently  efficacious  encoded  equally  familiar  fever  formyl  fpr2  isoforms  mediterranean  mobilization  modulating  predominant  preference  robust  saa1  selectivity  tlr2  tlr4lps  toll 
admission  atherosclerosis  believed  concentration  consequence  controls  explain  explanation  explicable  fifth  genotypes  hdl  immediately  infarction  mi  microg  min  ml  myocardial  nmol  occured  paraoxonase  pon1  reside  risen  subfraction  subgroups  thrombolytic  variation 
Tagcloud (Intersection) ?