PELI1 and RIPK1

  • Number of citations of the paper that reports this interaction (PMID 19734906)
  • 33
  • Data Source:
  • BioGRID (enzymatic study)

PELI1

RIPK1

Gene Name pellino E3 ubiquitin protein ligase 1 receptor (TNFRSF)-interacting serine-threonine kinase 1
Image No pdb structure
Gene Ontology Annotations Cellular Component
Molecular Function
Biological Process
Pathways
Drugs
Diseases
GWAS
Protein-Protein Interactions 17 interactors: BCL6 IKBKB IRAK1 IRAK2 IRAK4 RIPK1 SRPK1 SRPK2 TBK1 TRIP13 UBC UBE2D1 UBE2D2 UBE2I UBE2N UBE2V1 VAC14 45 interactors: BIRC2 BIRC3 CASP8 CCDC50 CDC42BPB CFLAR CRADD EGFR FADD FAS HSP90AA1 IKBKB IKBKG ITCH MAP3K14 MAP3K3 MAP3K8 NFKB1 NUMBL OTUD1 PELI1 PTK2 RIPK3 RNF11 RNF216 SQSTM1 TAX1BP1 TICAM1 TNF TNFAIP3 TNFRSF10A TNFRSF10B TNFRSF1A TRADD TRAF1 TRAF2 TRAF3 TRAF5 TRIM38 TRPC4AP UBE2N USP2 XIAP YOD1 ZFAND5
Entrez ID 57162 8737
HPRD ID 17834 04583
Ensembl ID ENSG00000197329 ENSG00000137275
Uniprot IDs Q53T26 Q96FA3 Q13546
PDB IDs 4ITH 4ITI 4ITJ
Enriched GO Terms of Interacting Partners?
Tagcloud ?
ablation  abundantly  autoimmune  chemokines  cns  competent  deficient  e3  eae  encephalomyelitis  induction  ligase  lymphoid  mediates  mediator  microglia  microglial  mitogen  nervous  neuroinflammation  notably  organs  proinflammatory  promotes  restores  sclerosis  tlr  toll  traf3 
allowed  autocrine  back  deficient  endoplasmic  er  instead  interacting  intrinsic  kills  l929  lineage  lymphotoxin  mlkl  necroptosis  necrosis  opportunities  pathologies  protect  remarkably  repression  rescue  reticulum  ripk3  stress  stressors  switch  tnfr1  unresolved 
Tagcloud (Difference) ?
ablation  abundantly  autoimmune  chemokines  cns  competent  e3  eae  encephalomyelitis  induction  ligase  lymphoid  mediates  mediator  microglia  microglial  mitogen  nervous  neuroinflammation  notably  organs  proinflammatory  promotes  restores  sclerosis  tlr  toll  traf3 
allowed  autocrine  back  endoplasmic  er  instead  interacting  intrinsic  kills  l929  lineage  lymphotoxin  mlkl  necroptosis  necrosis  opportunities  pathologies  protect  remarkably  repression  rescue  reticulum  ripk3  stress  stressors  switch  tnfr1  unresolved 
Tagcloud (Intersection) ?
deficient