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SLC5A6 and GALNT6
Number of citations of the paper that reports this interaction (PMID
21988832
)
14
Data Source:
BioGRID
(two hybrid)
SLC5A6
GALNT6
Gene Name
solute carrier family 5 (sodium/multivitamin and iodide cotransporter), member 6
polypeptide N-acetylgalactosaminyltransferase 6
Image
No pdb structure
No pdb structure
Gene Ontology Annotations
Cellular Component
Plasma Membrane
Integral Component Of Plasma Membrane
Vesicle Membrane
Membrane
Brush Border Membrane
Golgi Membrane
Golgi Apparatus
Integral Component Of Membrane
Perinuclear Region Of Cytoplasm
Molecular Function
Sodium-dependent Multivitamin Transmembrane Transporter Activity
Polypeptide N-acetylgalactosaminyltransferase Activity
Carbohydrate Binding
Metal Ion Binding
Biological Process
Vitamin Metabolic Process
Water-soluble Vitamin Metabolic Process
Biotin Metabolic Process
Transport
Sodium Ion Transport
Biotin Transport
Pantothenate Transmembrane Transport
Pantothenate Metabolic Process
Small Molecule Metabolic Process
Transmembrane Transport
Protein O-linked Glycosylation
O-glycan Processing
Post-translational Protein Modification
Cellular Protein Metabolic Process
Pathways
Metabolism of vitamins and cofactors
Defective CD320 causes methylmalonic aciduria
Defective LMBRD1 causes methylmalonic aciduria and homocystinuria type cblF
Defective BTD causes biotidinase deficiency
Defective MMACHC causes methylmalonic aciduria and homocystinuria type cblC
Defective MTR causes methylmalonic aciduria and homocystinuria type cblG
SLC-mediated transmembrane transport
Defective MTRR causes methylmalonic aciduria and homocystinuria type cblE
Defective AMN causes hereditary megaloblastic anemia 1
Transport of vitamins, nucleosides, and related molecules
Defects in cobalamin (B12) metabolism
Defective GIF causes intrinsic factor deficiency
Vitamin B5 (pantothenate) metabolism
Defective HLCS causes multiple carboxylase deficiency
Defective MMAB causes methylmalonic aciduria type cblB
Defective MMADHC causes methylmalonic aciduria and homocystinuria type cblD
Defective MMAA causes methylmalonic aciduria type cblA
Defective CUBN causes hereditary megaloblastic anemia 1
Defective MUT causes methylmalonic aciduria mut type
Metabolism of water-soluble vitamins and cofactors
Defects in biotin (Btn) metabolism
Defective TCN2 causes hereditary megaloblastic anemia
Biotin transport and metabolism
Defects in vitamin and cofactor metabolism
Post-translational protein modification
O-linked glycosylation of mucins
O-linked glycosylation
Drugs
Biotin
Lipoic Acid
Diseases
GWAS
Protein-Protein Interactions
2 interactors:
ERBB2IP
GALNT6
5 interactors:
ALDOA
FN1
SLC5A6
TAF9
TMEM9
Entrez ID
8884
11226
HPRD ID
04934
05512
Ensembl ID
ENSG00000138074
ENSG00000139629
Uniprot IDs
Q9HD19
Q9Y289
Q8NCL4
PDB IDs
Enriched GO Terms of Interacting Partners
?
Basal Protein Localization
Establishment Or Maintenance Of Epithelial Cell Apical/basal Polarity
Asymmetric Protein Localization
Establishment Or Maintenance Of Apical/basal Cell Polarity
Intermediate Filament Cytoskeleton Organization
Intermediate Filament-based Process
O-glycan Processing
Platelet Degranulation
Pantothenate Transmembrane Transport
Biotin Transport
Positive Regulation Of Cell Growth
Regulation Of Cell Shape
Platelet Activation
Fructose 1,6-bisphosphate Metabolic Process
Calcium-independent Cell-matrix Adhesion
Positive Regulation Of Growth
Exocytosis
Regulation Of Cell Growth
Pantothenate Metabolic Process
Integrin Activation
Biotin Metabolic Process
Negative Regulation Of Intrinsic Apoptotic Signaling Pathway In Response To DNA Damage By P53 Class Mediator
Vitamin Transmembrane Transport
Blood Coagulation
Hemostasis
Negative Regulation Of Intrinsic Apoptotic Signaling Pathway By P53 Class Mediator
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Tagcloud (Difference)
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Tagcloud (Intersection)
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