GALNT6 and SLC5A6

Number of citations of the paper that reports this interaction (PMID 21988832)
14

Data Source:
BioGRID (two hybrid)

		GALNT6	SLC5A6
Gene Name		polypeptide N-acetylgalactosaminyltransferase 6	solute carrier family 5 (sodium/multivitamin and iodide cotransporter), member 6
Image		No pdb structure	No pdb structure
Gene Ontology Annotations	Cellular Component	Golgi Membrane Golgi Apparatus Integral Component Of Membrane Perinuclear Region Of Cytoplasm	Plasma Membrane Integral Component Of Plasma Membrane Vesicle Membrane Membrane Brush Border Membrane
	Molecular Function	Polypeptide N-acetylgalactosaminyltransferase Activity Carbohydrate Binding Metal Ion Binding	Sodium-dependent Multivitamin Transmembrane Transporter Activity
	Biological Process	Protein O-linked Glycosylation O-glycan Processing Post-translational Protein Modification Cellular Protein Metabolic Process	Vitamin Metabolic Process Water-soluble Vitamin Metabolic Process Biotin Metabolic Process Transport Sodium Ion Transport Biotin Transport Pantothenate Transmembrane Transport Pantothenate Metabolic Process Small Molecule Metabolic Process Transmembrane Transport
Pathways		Post-translational protein modification O-linked glycosylation of mucins O-linked glycosylation	Metabolism of vitamins and cofactors Defective CD320 causes methylmalonic aciduria Defective LMBRD1 causes methylmalonic aciduria and homocystinuria type cblF Defective BTD causes biotidinase deficiency Defective MMACHC causes methylmalonic aciduria and homocystinuria type cblC Defective MTR causes methylmalonic aciduria and homocystinuria type cblG SLC-mediated transmembrane transport Defective MTRR causes methylmalonic aciduria and homocystinuria type cblE Defective AMN causes hereditary megaloblastic anemia 1 Transport of vitamins, nucleosides, and related molecules Defects in cobalamin (B12) metabolism Defective GIF causes intrinsic factor deficiency Vitamin B5 (pantothenate) metabolism Defective HLCS causes multiple carboxylase deficiency Defective MMAB causes methylmalonic aciduria type cblB Defective MMADHC causes methylmalonic aciduria and homocystinuria type cblD Defective MMAA causes methylmalonic aciduria type cblA Defective CUBN causes hereditary megaloblastic anemia 1 Defective MUT causes methylmalonic aciduria mut type Metabolism of water-soluble vitamins and cofactors Defects in biotin (Btn) metabolism Defective TCN2 causes hereditary megaloblastic anemia Biotin transport and metabolism Defects in vitamin and cofactor metabolism
Drugs			Biotin Lipoic Acid
Diseases
GWAS
Protein-Protein Interactions		5 interactors: ALDOA FN1 SLC5A6 TAF9 TMEM9	2 interactors: ERBB2IP GALNT6
Entrez ID		11226	8884
HPRD ID		05512	04934
Ensembl ID		ENSG00000139629	ENSG00000138074
Uniprot IDs		Q8NCL4	Q9HD19 Q9Y289
PDB IDs
Enriched GO Terms of Interacting Partners?		Platelet Degranulation Pantothenate Transmembrane Transport Biotin Transport Positive Regulation Of Cell Growth Regulation Of Cell Shape Platelet Activation Fructose 1,6-bisphosphate Metabolic Process Calcium-independent Cell-matrix Adhesion Positive Regulation Of Growth Exocytosis Regulation Of Cell Growth Pantothenate Metabolic Process Integrin Activation Biotin Metabolic Process Negative Regulation Of Intrinsic Apoptotic Signaling Pathway In Response To DNA Damage By P53 Class Mediator Vitamin Transmembrane Transport Blood Coagulation Hemostasis Negative Regulation Of Intrinsic Apoptotic Signaling Pathway By P53 Class Mediator	Basal Protein Localization Establishment Or Maintenance Of Epithelial Cell Apical/basal Polarity Asymmetric Protein Localization Establishment Or Maintenance Of Apical/basal Cell Polarity Intermediate Filament Cytoskeleton Organization Intermediate Filament-based Process O-glycan Processing
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