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B4GALT3 and SURF2
Number of citations of the paper that reports this interaction (PMID
16169070
)
531
Data Source:
HPRD
(two hybrid)
B4GALT3
SURF2
Gene Name
UDP-Gal:betaGlcNAc beta 1,4- galactosyltransferase, polypeptide 3
surfeit 2
Image
No pdb structure
No pdb structure
Gene Ontology Annotations
Cellular Component
Golgi Membrane
Cytoplasm
Golgi Apparatus
Integral Component Of Membrane
Golgi Cisterna Membrane
Extracellular Vesicular Exosome
Cellular_component
Nucleus
Nucleolus
Plasma Membrane
Molecular Function
Beta-N-acetylglucosaminylglycopeptide Beta-1,4-galactosyltransferase Activity
N-acetyllactosamine Synthase Activity
Galactosyltransferase Activity
Metal Ion Binding
Molecular_function
Biological Process
Carbohydrate Metabolic Process
Pathogenesis
Keratan Sulfate Biosynthetic Process
Protein N-linked Glycosylation Via Asparagine
Glycosaminoglycan Metabolic Process
Keratan Sulfate Metabolic Process
Post-translational Protein Modification
Cellular Protein Metabolic Process
Small Molecule Metabolic Process
Biological_process
Pathways
Keratan sulfate/keratin metabolism
MPS I - Hurler syndrome
MPS IX - Natowicz syndrome
Glycosaminoglycan metabolism
Defective MGAT2 causes MGAT2-CDG (CDG-2a)
N-Glycan antennae elongation
Defective CHST14 causes EDS, musculocontractural type
N-glycan antennae elongation in the medial/trans-Golgi
Myoclonic epilepsy of Lafora
MPS IIID - Sanfilippo syndrome D
Defective CHST6 causes MCDC1
Glycogen storage diseases
Keratan sulfate biosynthesis
Defective ALG2 causes ALG2-CDG (CDG-1i)
Diseases associated with glycosaminoglycan metabolism
Mucopolysaccharidoses
Defective ALG3 causes ALG3-CDG (CDG-1d)
Post-translational protein modification
Defective EXT2 causes exostoses 2
Asparagine N-linked glycosylation
MPS IV - Morquio syndrome A
Defective MPDU1 causes MPDU1-CDG (CDG-1f)
Metabolism of carbohydrates
Diseases associated with N-glycosylation of proteins
MPS VI - Maroteaux-Lamy syndrome
MPS IIIB - Sanfilippo syndrome B
Diseases of glycosylation
Defective B4GALT7 causes EDS, progeroid type
Defective ALG14 causes congenital myasthenic syndrome (ALG14-CMS)
Defective SLC26A2 causes chondrodysplasias
Transport to the Golgi and subsequent modification
Defective ALG1 causes ALG1-CDG (CDG-1k)
Defective MOGS causes MOGS-CDG (CDG-2b)
Defective B4GALT1 causes B4GALT1-CDG (CDG-2d)
Defective ALG9 causes ALG9-CDG (CDG-1l)
Defective PAPSS2 causes SEMD-PA
Defective MAN1B1 causes MRT15
MPS IIIA - Sanfilippo syndrome A
Defective ALG11 causes ALG11-CDG (CDG-1p)
MPS IIIC - Sanfilippo syndrome C
Defective DPAGT1 causes DPAGT1-CDG (CDG-1j) and CMSTA2
MPS II - Hunter syndrome
Defective CHST3 causes SEDCJD
Defective B3GAT3 causes JDSSDHD
Defective EXT1 causes exostoses 1, TRPS2 and CHDS
Defective B4GALT1 causes B4GALT1-CDG (CDG-2d)
Defective RFT1 causes RFT1-CDG (CDG-1n)
Defective ALG6 causes ALG6-CDG (CDG-1c)
Defective ALG8 causes ALG8-CDG (CDG-1h)
Defective ALG12 causes ALG12-CDG (CDG-1g)
Defective CHSY1 causes TPBS
MPS IV - Morquio syndrome B
MPS VII - Sly syndrome
Drugs
Diseases
GWAS
Activated partial thromboplastin time (
22703881
)
Protein-Protein Interactions
3 interactors:
ARL15
SAT1
SURF2
7 interactors:
APP
B4GALT3
NPTN
PLAUR
PRNP
RPL5
ZNF331
Entrez ID
8703
6835
HPRD ID
04929
15948
Ensembl ID
ENSG00000158850
ENSG00000148291
Uniprot IDs
A8K5Z0
O60512
Q15527
PDB IDs
Enriched GO Terms of Interacting Partners
?
Spermidine Acetylation
Putrescine Catabolic Process
Oxalate Transport
3'-phosphoadenosine 5'-phosphosulfate Biosynthetic Process
Polyamine Catabolic Process
Neurotransmitter Uptake
3'-phosphoadenosine 5'-phosphosulfate Metabolic Process
Polyamine Biosynthetic Process
Amine Biosynthetic Process
Polyamine Metabolic Process
Sulfate Transmembrane Transport
Sulfate Transport
Neutral Amino Acid Transport
Cellular Copper Ion Homeostasis
Copper Ion Homeostasis
Cellular Metal Ion Homeostasis
Cellular Cation Homeostasis
Cellular Ion Homeostasis
Metal Ion Homeostasis
Urokinase Plasminogen Activator Signaling Pathway
Cellular Chemical Homeostasis
Ion Homeostasis
Chemotaxis
Synaptic Growth At Neuromuscular Junction
Transition Metal Ion Homeostasis
Cellular Homeostasis
Collateral Sprouting In Absence Of Injury
Negative Regulation Of Calcineurin-NFAT Signaling Cascade
Collateral Sprouting
Axon Midline Choice Point Recognition
Post-translational Protein Modification
Chemical Homeostasis
Smooth Endoplasmic Reticulum Calcium Ion Homeostasis
Cellular Response To Copper Ion
Axon Choice Point Recognition
Negative Regulation Of Calcium-mediated Signaling
Regulation Of Protein Metabolic Process
Positive Regulation Of Long-term Neuronal Synaptic Plasticity
Learning Or Memory
Neuron Remodeling
Negative Regulation Of Interleukin-17 Production
Regulation Of Protein Phosphorylation
Regulation Of Calcineurin-NFAT Signaling Cascade
Cognition
Cellular Calcium Ion Homeostasis
Cellular Protein Metabolic Process
Calcium Ion Homeostasis
Attachment Of GPI Anchor To Protein
Negative Regulation Of Activated T Cell Proliferation
Positive Regulation Of Fibroblast Growth Factor Receptor Signaling Pathway
Locomotion
Regulation Of Phosphorylation
Axon Guidance
Negative Regulation Of Interleukin-2 Production
Homeostatic Process
Negative Regulation Of T Cell Receptor Signaling Pathway
Response To Oxidative Stress
Blood Coagulation
Hemostasis
Response To Copper Ion
Suckling Behavior
Regulation Of Phosphorus Metabolic Process
Movement Of Cell Or Subcellular Component
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Tagcloud (Difference)
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Tagcloud (Intersection)
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