CDKN2A and GGA1 |
cyclin-dependent kinase inhibitor 2A |
golgi-associated, gamma adaptin ear containing, ARF binding protein 1 |
- Cyclin D associated events in G1
- Oxidative Stress Induced Senescence
- G1 Phase
- Cellular Senescence
- Senescence-Associated Secretory Phenotype (SASP)
- Oncogene Induced Senescence
- Mitotic G1-G1/S phases
- Cell Cycle, Mitotic
- Oxidative Stress Induced Senescence
- Cellular Senescence
- Oncogene Induced Senescence
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CDKN2A and PCNA |
cyclin-dependent kinase inhibitor 2A |
proliferating cell nuclear antigen |
- Cyclin D associated events in G1
- Oxidative Stress Induced Senescence
- G1 Phase
- Cellular Senescence
- Senescence-Associated Secretory Phenotype (SASP)
- Oncogene Induced Senescence
- Mitotic G1-G1/S phases
- Cell Cycle, Mitotic
- Oxidative Stress Induced Senescence
- Cellular Senescence
- Oncogene Induced Senescence
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- Nucleotide Excision Repair
- Gap-filling DNA repair synthesis and ligation in TC-NER
- Extension of Telomeres
- Repair synthesis for gap-filling by DNA polymerase in TC-NER
- Leading Strand Synthesis
- Mismatch repair (MMR) directed by MSH2:MSH6 (MutSalpha)
- DNA strand elongation
- Mismatch Repair
- Polymerase switching
- Telomere Maintenance
- G1/S Transition
- Removal of DNA patch containing abasic residue
- G1/S-Specific Transcription
- Resolution of Abasic Sites (AP sites)
- Mitotic G1-G1/S phases
- Removal of the Flap Intermediate from the C-strand
- Base Excision Repair
- E2F mediated regulation of DNA replication
- Polymerase switching on the C-strand of the telomere
- Chromosome Maintenance
- Synthesis of DNA
- Lagging Strand Synthesis
- Processive synthesis on the C-strand of the telomere
- Processive synthesis on the lagging strand
- G0 and Early G1
- Telomere C-strand (Lagging Strand) Synthesis
- S Phase
- Repair synthesis of patch ~27-30 bases long by DNA polymerase
- Cell Cycle, Mitotic
- Gap-filling DNA repair synthesis and ligation in GG-NER
- Removal of the Flap Intermediate
- Global Genomic NER (GG-NER)
- Mismatch repair (MMR) directed by MSH2:MSH3 (MutSbeta)
- Resolution of AP sites via the multiple-nucleotide patch replacement pathway
- Transcription-coupled NER (TC-NER)
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CDKN2A and EPHA3 |
cyclin-dependent kinase inhibitor 2A |
EPH receptor A3 |
- Cyclin D associated events in G1
- Oxidative Stress Induced Senescence
- G1 Phase
- Cellular Senescence
- Senescence-Associated Secretory Phenotype (SASP)
- Oncogene Induced Senescence
- Mitotic G1-G1/S phases
- Cell Cycle, Mitotic
- Oxidative Stress Induced Senescence
- Cellular Senescence
- Oncogene Induced Senescence
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- EPHA-mediated growth cone collapse
- EPH-ephrin mediated repulsion of cells
- Axon guidance
- EPH-Ephrin signaling
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CDKN2A and TPI1 |
cyclin-dependent kinase inhibitor 2A |
triosephosphate isomerase 1 |
- Cyclin D associated events in G1
- Oxidative Stress Induced Senescence
- G1 Phase
- Cellular Senescence
- Senescence-Associated Secretory Phenotype (SASP)
- Oncogene Induced Senescence
- Mitotic G1-G1/S phases
- Cell Cycle, Mitotic
- Oxidative Stress Induced Senescence
- Cellular Senescence
- Oncogene Induced Senescence
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- Gluconeogenesis
- Glucose metabolism
- Myoclonic epilepsy of Lafora
- Glycogen storage diseases
- Glycolysis
- Metabolism of carbohydrates
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CDKN2B and TGFB1I1 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
transforming growth factor beta 1 induced transcript 1 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDKN2B and IKBKAP |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
inhibitor of kappa light polypeptide gene enhancer in B-cells, kinase complex-associated protein |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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- Chromatin modifying enzymes
- Chromatin organization
- HATs acetylate histones
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CDKN2B and NIF3L1 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
NIF3 NGG1 interacting factor 3-like 1 (S. cerevisiae) |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDKN2B and CCDC90B |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
coiled-coil domain containing 90B |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDKN2B and ARPC3 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
actin related protein 2/3 complex, subunit 3, 21kDa |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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- EPHB-mediated forward signaling
- Axon guidance
- EPH-Ephrin signaling
- Fcgamma receptor (FCGR) dependent phagocytosis
- Regulation of actin dynamics for phagocytic cup formation
- Innate Immune System
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CDKN2B and TRAF1 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
TNF receptor-associated factor 1 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDKN2B and MAGEA11 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
melanoma antigen family A, 11 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDKN2B and ISL1 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
ISL LIM homeobox 1 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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- Peptide hormone metabolism
- Synthesis, secretion, and inactivation of Glucose-dependent Insulinotropic Polypeptide (GIP)
- Incretin synthesis, secretion, and inactivation
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CDKN2B and KIAA1377 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
KIAA1377 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDKN2B and ZBTB17 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
zinc finger and BTB domain containing 17 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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- IRE1alpha activates chaperones
- XBP1(S) activates chaperone genes
- Unfolded Protein Response (UPR)
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CDKN2B and CCDC33 |
cyclin-dependent kinase inhibitor 2B (p15, inhibits CDK4) |
coiled-coil domain containing 33 |
- Loss of Function of TGFBR2 in Cancer
- SMAD2/3 MH2 Domain Mutants in Cancer
- Cellular Senescence
- Oncogene Induced Senescence
- TGFBR1 LBD Mutants in Cancer
- SMAD2/SMAD3:SMAD4 heterotrimer regulates transcription
- Transcriptional activity of SMAD2/SMAD3:SMAD4 heterotrimer
- Generic Transcription Pathway
- Mitotic G1-G1/S phases
- TGFBR2 MSI Frameshift Mutants in Cancer
- SMAD2/3 Phosphorylation Motif Mutants in Cancer
- Loss of Function of SMAD2/3 in Cancer
- TGFBR2 Kinase Domain Mutants in Cancer
- Loss of Function of SMAD4 in Cancer
- G1 Phase
- Senescence-Associated Secretory Phenotype (SASP)
- TGFBR1 KD Mutants in Cancer
- Cell Cycle, Mitotic
- Loss of Function of TGFBR1 in Cancer
- Cyclin D associated events in G1
- Signaling by TGF-beta Receptor Complex in Cancer
- Signaling by TGF-beta Receptor Complex
- Oxidative Stress Induced Senescence
- SMAD4 MH2 Domain Mutants in Cancer
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CDX2 and HNF1A |
caudal type homeobox 2 |
HNF1 homeobox A |
- Peptide hormone metabolism
- Synthesis, secretion, and inactivation of Glucagon-like Peptide-1 (GLP-1)
- Incretin synthesis, secretion, and inactivation
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- Regulation of beta-cell development
- Regulation of gene expression in beta cells
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CEBPA and HNF1A |
CCAAT/enhancer binding protein (C/EBP), alpha |
HNF1 homeobox A |
- Transcriptional regulation of white adipocyte differentiation
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- Regulation of beta-cell development
- Regulation of gene expression in beta cells
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CHN2 and ERBB3 |
chimerin 2 |
erb-b2 receptor tyrosine kinase 3 |
- Rho GTPase cycle
- Signaling by Rho GTPases
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CHUK and PRKCQ |
conserved helix-loop-helix ubiquitous kinase |
protein kinase C, theta |
- Signaling by the B Cell Receptor (BCR)
- Signaling by FGFR in disease
- Signaling by EGFRvIII in Cancer
- AKT phosphorylates targets in the cytosol
- RIP-mediated NFkB activation via ZBP1
- IRAK1 recruits IKK complex
- Signaling by SCF-KIT
- Toll Like Receptor TLR1:TLR2 Cascade
- DAP12 signaling
- Downstream signaling events of B Cell Receptor (BCR)
- FCERI mediated NF-kB activation
- PI3K/AKT activation
- Toll Like Receptor 5 (TLR5) Cascade
- PI-3K cascade
- NOD1/2 Signaling Pathway
- MyD88 dependent cascade initiated on endosome
- Toll Like Receptor 9 (TLR9) Cascade
- IRAK1 recruits IKK complex
- Signaling by PDGF
- DAP12 interactions
- GAB1 signalosome
- TRIF-mediated TLR3/TLR4 signaling
- Signaling by ERBB4
- Role of LAT2/NTAL/LAB on calcium mobilization
- Constitutive PI3K/AKT Signaling in Cancer
- PI3K events in ERBB4 signaling
- Toll Like Receptor 2 (TLR2) Cascade
- Signaling by ERBB2
- Signaling by EGFR
- Signaling by Interleukins
- TCR signaling
- Downstream signal transduction
- Toll Like Receptor 3 (TLR3) Cascade
- Toll Like Receptor 4 (TLR4) Cascade
- Signaling by EGFR in Cancer
- Fc epsilon receptor (FCERI) signaling
- Interleukin-1 signaling
- PI3K/AKT Signaling in Cancer
- TAK1 activates NFkB by phosphorylation and activation of IKKs complex
- Adaptive Immune System
- Downstream TCR signaling
- PIP3 activates AKT signaling
- Toll Like Receptor 7/8 (TLR7/8) Cascade
- Toll Like Receptor TLR6:TLR2 Cascade
- Activated TLR4 signalling
- MyD88 cascade initiated on plasma membrane
- ZBP1(DAI) mediated induction of type I IFNs
- PI3K events in ERBB2 signaling
- Downstream signaling of activated FGFR
- TRAF6 mediated induction of NFkB and MAP kinases upon TLR7/8 or 9 activation
- MyD88:Mal cascade initiated on plasma membrane
- Innate Immune System
- Signalling by NGF
- IKK complex recruitment mediated by RIP1
- Cytosolic sensors of pathogen-associated DNA
- Cytokine Signaling in Immune system
- Signaling by Ligand-Responsive EGFR Variants in Cancer
- NGF signalling via TRKA from the plasma membrane
- Signaling by Overexpressed Wild-Type EGFR in Cancer
- MyD88-independent cascade
- Signaling by FGFR
- RIG-I/MDA5 mediated induction of IFN-alpha/beta pathways
- Toll-Like Receptors Cascades
- Toll Like Receptor 10 (TLR10) Cascade
- Nucleotide-binding domain, leucine rich repeat containing receptor (NLR) signaling pathways
- Activation of NF-kappaB in B cells
- TRAF6 mediated NF-kB activation
- NF-kB activation through FADD/RIP-1 pathway mediated by caspase-8 and -10
- IRAK1 recruits IKK complex upon TLR7/8 or 9 stimulation
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- G alpha (q) signalling events
- Signaling by GPCR
- Downstream TCR signaling
- Axon guidance
- Effects of PIP2 hydrolysis
- FCERI mediated NF-kB activation
- Inactivation, recovery and regulation of the phototransduction cascade
- Gastrin-CREB signalling pathway via PKC and MAPK
- The phototransduction cascade
- Programmed Cell Death
- G alpha (z) signalling events
- Netrin-1 signaling
- Innate Immune System
- Apoptotic cleavage of cellular proteins
- Apoptotic execution phase
- Diseases associated with visual transduction
- G alpha (q) signalling events
- GPCR downstream signaling
- TCR signaling
- Visual phototransduction
- Fc epsilon receptor (FCERI) signaling
- Platelet activation, signaling and aggregation
- Adaptive Immune System
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- Staurosporine
- Phosphonothreonine
- Phosphonoserine
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CISH and PRKCQ |
cytokine inducible SH2-containing protein |
protein kinase C, theta |
- Cytokine Signaling in Immune system
- Growth hormone receptor signaling
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- G alpha (q) signalling events
- Signaling by GPCR
- Downstream TCR signaling
- Axon guidance
- Effects of PIP2 hydrolysis
- FCERI mediated NF-kB activation
- Inactivation, recovery and regulation of the phototransduction cascade
- Gastrin-CREB signalling pathway via PKC and MAPK
- The phototransduction cascade
- Programmed Cell Death
- G alpha (z) signalling events
- Netrin-1 signaling
- Innate Immune System
- Apoptotic cleavage of cellular proteins
- Apoptotic execution phase
- Diseases associated with visual transduction
- G alpha (q) signalling events
- GPCR downstream signaling
- TCR signaling
- Visual phototransduction
- Fc epsilon receptor (FCERI) signaling
- Platelet activation, signaling and aggregation
- Adaptive Immune System
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- Staurosporine
- Phosphonothreonine
- Phosphonoserine
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